How Long Does Meth Stay In Your System?

Key Points (2026)

• • Urine tests (most common): Detect meth for 2-4 days in occasional users, extending to 5-7+ days in chronic users. This is what most workplaces use.
  • Hair tests (longest detection): Can identify meth use for up to 90 days after the last use, making them valuable for pattern identification.
  • Blood and saliva: Only detect meth for 1-4 days, which is why they’re less common for workplace screening despite being more direct measures.
  • Half-life and complete clearance: Meth has a 10-12 hour half-life, meaning roughly 50-75 hours (2-3 days) for the drug itself to clear bloodstream completely, though urine metabolites persist longer.
  • Chronic use accumulation: People who use meth regularly develop longer detection windows because the drug accumulates in fatty tissues and gets slowly released back into circulation over time.
  • Withdrawal doesn’t match detection: While meth becomes undetectable in days, withdrawal symptoms – particularly the psychological ones – persist for weeks to months as the brain recalibrates.

How Long Does Meth Stay In Your System – What Drug Tests Actually Measure – And What They Miss

Understanding drug testing starts with understanding that different tests measure different things at different times. A negative drug test doesn’t mean the person is “clean” in any meaningful recovery sense – it just means the drug isn’t present in detectable amounts in that particular biological sample. This distinction matters because it shapes how people interpret test results and what they actually tell us about recovery status.

Urine Testing – The Workplace Standard

Urine testing dominates workplace and legal drug screening because it’s inexpensive, non-invasive, and provides a reasonable window into recent use. The standard federal cutoff is 500 ng/mL for initial screening, with confirmation at 250 ng/mL using gas chromatography-mass spectrometry (GC-MS). For someone who uses meth C10H15N once, the drug typically appears in urine within 3-6 hours and remains detectable for 2-4 days. Someone using 2-3 times weekly gets into different territory – those detection windows extend to 4-7 days because the body hasn’t fully cleared the previous dose before new drug arrives. For daily or near-daily users, urine can stay positive for 7+ days. The cutoff levels exist specifically to reduce false positives from things like decongestants containing pseudoephedrine, though confirmation testing eliminates most of these issues.

Blood Testing – More Accurate But Shorter Window

Blood tests are more precise measures of active drug presence because they’re measuring actual meth molecules rather than metabolites, but they only detect the drug for 1-3 days – typically 24-48 hours. This makes them less practical for workplace screening but more useful in emergency medical situations where knowing recent use matters for treatment decisions. Medical providers use blood tests when they need to confirm current intoxication rather than past use patterns.

Saliva/Oral Fluid – The Emerging Option

Saliva testing is becoming more common because it’s hard to cheat (you can’t substitute someone else’s saliva easily), provides on-site results, and is non-invasive. Detection ranges from minutes after use up to 1-4 days depending on individual metabolism and how frequently someone has been using. The window varies more than other methods based on timing – if someone tests right after using, saliva is extremely sensitive. Testing days later becomes less reliable.

Hair Testing – Pattern Recognition Over Recent Use

Hair testing reveals patterns of use rather than recent use, detecting meth for approximately 90 days (3 months) after use stops. A standard 1.5-inch hair sample represents roughly 90 days of history. This makes hair testing particularly useful in custody evaluations, treatment program entry assessments, and situations where identifying long-term use patterns matters more than detecting recent use. The test doesn’t measure how much someone used, just whether use occurred during that timeframe. One important limitation: factors like hair color, certain hair products, and individual hair growth rates can affect sensitivity.

How Your Body Actually Processes Methamphetamine

The detection timeline ultimately comes down to how your body breaks down and eliminates meth. Understanding this process explains why some people test positive longer than others and why the timeline isn’t simple or universal.

The Half-Life Concept and What It Actually Means

Methamphetamine has a half-life of approximately 10-12 hours in most people. This means that 10-12 hours after use, half of the ingested dose remains in your body. Twelve hours later, half of that half remains (25% of original). Continue this pattern and after roughly 50-75 hours (about 2-3 days), the original amount has been reduced to negligible levels in the bloodstream. However – and this is important – negligible in the bloodstream doesn’t mean undetectable in urine, because the kidneys filter metabolites at different rates than the bloodstream clears the drug itself. This is why someone might have a negative blood test but a positive urine test on the same day.

Where Meth Goes First – Organ Distribution

When methamphetamine enters your bloodstream, it doesn’t distribute evenly. It accumulates fastest in organs with high blood flow and lipid content. Studies tracking meth distribution show the lungs contain 22-31% of the dose, the liver about 23%, and the kidneys 7%. The important detail here is speed – the heart and lungs clear meth fastest, achieving half-clearance in just 7-16 minutes. The brain, conversely, clears meth extremely slowly with half-clearance occurring after 75+ minutes or longer. This asymmetry explains why cardiovascular effects are most intense immediately after use (heart and lungs are flooding with meth) while behavioral effects persist after the dose wears off (brain is clearing it slowly).

Liver Metabolism – The Enzyme System

The liver does the actual work of breaking down methamphetamine. An enzyme called cytochrome P450 2D6 (CYP2D6) converts meth into two main breakdown products: para-hydroxymethamphetamine and amphetamine. About 37-54% of ingested meth gets excreted unchanged in urine, while 6-7% gets converted to amphetamine and then excreted, with most elimination occurring within 72 hours. Here’s where individual variation enters: genetic differences in the CYP2D6 enzyme mean some people metabolize meth significantly faster or slower than others. Someone with a “rapid metabolizer” version of this enzyme clears meth quicker; someone with a “poor metabolizer” version takes longer. These genetic differences can account for meaningful variations in detection windows between people with identical use patterns.

Kidney Clearance and Urine pH

Once the liver processes meth, the kidneys filter the metabolites from blood and excrete them in urine. Approximately 10% of an injected meth dose appears in urine within 90 minutes, with most metabolites cleared within 72 hours. An interesting wrinkle: because methamphetamine is a weak base, acidic urine actually traps and concentrates meth metabolites, potentially extending how long they remain detectable. This is sometimes why people with certain diets or underlying metabolic conditions show longer detection windows – their urine pH naturally runs more acidic. The flip side: claims that drinking water, taking baking soda, or other dietary interventions can significantly alter urine pH and speed clearance are largely myths. The body maintains relatively stable urine pH through homeostasis, and these interventions don’t produce clinically meaningful changes.

Why Timelines Vary – Individual Factors That Matter

Detection windows are never uniform because multiple factors specific to each person influence how quickly they metabolize and eliminate meth. Understanding what actually affects your personal timeline is more useful than memorizing generic numbers.

How Often You Use Matters Most

Frequency of use is the single biggest determinant of detection time. Someone using meth once or twice has it detectable for 2-4 days in urine. Someone using 2-3 times per week enters different territory – detection typically extends to 4-7 days because the body doesn’t fully clear one dose before the next arrives. For daily users or those in binge patterns (using multiple times per day over several days), detection can persist 7+ days. The reason is straightforward: your liver and kidneys have a maximum processing capacity. Load them with more drug than they can process, and a backlog develops where metabolites continue being cleared for extended periods.

Route of Administration – Smoking vs. Snorting vs. Swallowing

How meth enters your body does affect timing, but less dramatically than people often think. Smoking or injecting delivers meth directly into the bloodstream, producing rapid onset and theoretically faster clearance since peak concentrations are reached and eliminated more quickly. Snorting produces intermediate absorption, while swallowing produces slower absorption as the drug travels through the digestive system and gets partially degraded by stomach acid. Despite these differences in onset, the total elimination timeline doesn’t vary dramatically – mainly the timing of when peak effects occur. A person who smokes meth and a person who snorts the same amount will both likely test positive for similar durations, though the smoker feels effects sooner.

Purity and What Else Is in the Drug

Street methamphetamine purity ranges from 60-90% depending on the source and how many times it’s been adulterated. Purer meth might be metabolized slightly faster than adulterated meth because there’s less non-meth substance for the body to process. When meth is cut with other chemicals, those contaminants introduce additional compounds that your liver and kidneys must process, potentially extending the overall clearance timeline. This is one reason why detection windows can vary significantly even among people using similar amounts and frequencies – they’re often using different chemical mixtures.

Body Weight and Composition

Methamphetamine is lipophilic, meaning it dissolves readily in fat. This matters because a person with higher body fat percentage will have longer detection windows than a lean person using identical amounts and patterns. The drug literally accumulates in fatty tissue, creating a reservoir that slowly releases back into circulation over time. Someone who is obese might test positive 1-2 days longer than someone lean with the same use history. Age also plays a role – younger people tend to metabolize meth faster than older individuals, though the difference is usually modest.

Liver and Kidney Function

Any compromise to liver or kidney function directly extends detection time because these organs do the actual work of processing and eliminating meth. Someone with liver disease, hepatitis, or cirrhosis processes meth more slowly because the CYP2D6 enzyme doesn’t function optimally. Similarly, kidney disease or kidney dysfunction impairs excretion of metabolites, keeping them in the body longer. Certain medications that inhibit CYP2D6 (some antidepressants, antipsychotics, and other drugs) can also slow meth metabolism.

Chronic Use Creates an Accumulation Problem

This is worth understanding specifically because it affects people in active addiction more than casual users. With repeated use, meth doesn’t fully clear between doses – it accumulates in fatty tissues including the brain, organs, and subcutaneous fat. This creates a biological reservoir where drug continues being mobilized and eliminated even after someone stops using. A chronic user sometimes reports surprisingly prolonged detection despite thinking they should test clean after a certain timeframe – that’s the reservoir effect as stored drug gets gradually released back into circulation. One study found that approximately one-third of ingested meth remains in the body unchanged and gets re-released gradually, extending the clearance process well beyond what the simple half-life would suggest.

The Acute Phase – What Actually Happens During Use

Understanding the acute effects of meth provides context for why detection windows exist and what’s happening in the body during those first hours and days. The acute phase looks very different from the withdrawal phase that follows.

The Rush and Peak Effects

When meth hits the brain, it floods dopamine pathways with amounts of neurotransmitter the brain doesn’t normally experience. If someone smokes or injects, this rush occurs within seconds, creating intense euphoria, confidence, and heightened alertness that lasts 5-30 minutes depending on route and individual sensitivity. During this rush, heart rate and blood pressure spike dramatically, pupils dilate, and body temperature begins rising. Simultaneously, the liver starts processing the drug and the kidneys begin accumulating metabolites. Interestingly, at the peak of the rush when effects feel most intense, the amount of drug detectable in urine is still relatively low – it takes time for metabolites to accumulate to detectable levels.

The Extended High and Neurological Persistence

The rush transitions into a more sustained high lasting 4-16 hours (with smoking producing shorter duration, oral use producing longer). During this extended phase, the intense euphoria fades into hyperactivity, rapid thinking, decreased appetite, and wakefulness. Here’s an important point: the behavioral and neurological effects persist far longer than the drug remains at high concentrations in the bloodstream. Someone might feel completely normal, think the meth has “worn off,” but still test positive on a urine drug test 24-48 hours later because metabolites remain accumulating in urine even as brain concentration drops.

Cardiovascular Effects and the Danger Window

Meth’s cardiovascular effects represent the major acute toxicity risk. The drug produces sympathomimetic effects (mimicking adrenaline) that elevate blood pressure, heart rate, and respiration for hours after use. Sudden heart attack, stroke, and dangerous arrhythmias occur during or shortly after use, not during withdrawal or days later. The brain clears meth slowly (over 75+ minutes), meaning cognitive and behavioral effects persist as an aftereffect even when the drug has mostly cleared the rest of the body. This is why paranoia, agitation, and poor judgment can persist for hours after someone feels the drug “wearing off.”

Chronic Use and Brain Changes – Why Detection Timeline Matters Less Than Brain Recovery

For people using meth regularly, the picture becomes far more complex than detection timelines suggest. Chronic use creates persistent brain changes that continue well after the drug becomes undetectable in any test.

Dopamine System Dysregulation

Methamphetamine forces dopamine neurons to release massive amounts of dopamine far exceeding normal physiological levels. This extreme stimulation damages dopamine-producing neurons through multiple mechanisms: the drug generates toxic reactive oxygen species (free radicals), promotes inflammation, disrupts mitochondrial function, and activates neuronal death pathways. With chronic use, dopamine neurons gradually lose capacity to produce and release dopamine, resulting in decreased dopamine levels throughout the brain even months after someone stops using. Research comparing chronic meth users to healthy controls shows significant reductions in dopamine transporter density, dopamine D2 receptor availability, and overall dopamine function – changes that persist for extended periods after cessation.

Structural Brain Changes

Brain imaging reveals that chronic meth users have reduced gray matter volume in critical regions: the prefrontal cortex (decision-making and impulse control), anterior cingulate (emotional regulation), striatum (reward processing), and hippocampus (memory). White matter abnormalities also appear, indicating damage to neural connections between brain regions. These aren’t temporary changes – they represent actual neuronal death and reduced tissue volume that takes months to years to recover, if complete recovery occurs at all.

Cognitive Impairments in Chronic Users

The structural changes correlate with measurable cognitive deficits. Chronic meth users show significant impairments in episodic memory (the ability to remember specific past events), executive function (planning, decision-making, impulse control), attention, and processing speed. These aren’t subtle differences – they’re often clinically significant enough to interfere with work, education, and daily functioning. Importantly, these cognitive problems persist well after meth becomes undetectable through standard drug testing, sometimes lingering for months or years.

Neuroinflammation and Oxidative Damage

The damage mechanism involves activation of brain immune cells (microglia) that produce inflammatory cytokines like TNF-alpha and IL-1, creating chronic inflammation that perpetuates neuronal damage. Simultaneously, meth-induced free radicals damage mitochondria – the cellular energy factories – disrupting the brain’s ability to produce energy and maintain neuronal function. This inflammatory cascade continues even after meth becomes undetectable, which is why withdrawing individuals can experience neurological symptoms for weeks when the drug is no longer in their system.

Withdrawal Timeline – Where the Real Challenge Begins

Methamphetamine becomes undetectable through standard drug testing in 2-4 days. But this is precisely when the real challenge for people in recovery actually begins. Understanding withdrawal helps explain why detection timelines are almost irrelevant to actual recovery outcomes.

The Crash – 24-48 Hours After Last Use

When someone stops using meth after regular use, the crash hits hard. The brain suddenly lacks the dopamine flood it had adapted to, triggering a dramatic neurochemical shift from overstimulation to profound depletion. The crash typically peaks 24-48 hours after the last use and features overwhelming fatigue and sleepiness as the body desperately tries to recover from extended wakefulness (meth suppresses sleep for days during active use). Accompanying this physical crash is severe depression and anhedonia – the inability to feel pleasure or motivation from anything, even things that normally bring joy. Many people also experience anxiety, agitation, or paranoia during the crash phase. Physically, users report aches, headaches, and intense appetite rebound as their body signals depletion. This crash phase is profoundly uncomfortable – which is precisely why so many people relapse during this window. The discomfort can feel unbearable, and returning to meth provides immediate (though temporary) relief.

Acute Withdrawal – Days 3 Through 14

After the initial crash subsides, users enter acute withdrawal proper. Paradoxically, this is when something gets worse: cravings intensify dramatically to their peak level around day 5-7. This is the highest-risk period for relapse because cravings combined with residual depression and anhedonia create powerful internal pressure to use again. Physical withdrawal symptoms during this phase include sleep disturbance (insomnia alternating with oversleeping), persistent headaches, appetite changes (extreme hunger followed by nausea), and muscle aches. Psychologically, people experience agitation, irritability, difficulty concentrating, persistent anxiety, and continued depression. Some people experience residual paranoia or visual disturbances during acute withdrawal as the brain continues recalibrating its neurotransmitter systems.

Protracted Withdrawal – Weeks 2 Through 12 and Beyond

Around day 14, the acute phase transitions into protracted withdrawal syndrome (PAWS), where physical symptoms largely resolve but psychological symptoms become predominant and variable. This phase can persist for weeks to months with gradually improving symptoms. During PAWS, people experience anhedonia (persistent inability to feel pleasure or motivation), depression that can be severe and lingering, anxiety including panic attacks, difficulty concentrating and memory problems, and ongoing sleep disturbances. Cravings continue during this phase though typically with reduced intensity compared to acute withdrawal. A critical point: PAWS symptoms can appear inconsistent and cyclical – good days alternating with difficult days – which undermines confidence in recovery and increases relapse risk. The neurobiological basis for PAWS reflects the slow restoration of dopamine system function; dopamine transporter density and dopamine D2 receptor availability remain significantly reduced for months to years after stopping meth use, meaning the brain’s reward system is literally still damaged and recovering.

Variables Affecting Withdrawal Severity

Not everyone experiences withdrawal identically. Longer histories of heavy meth use produce more severe and extended withdrawal than occasional use. Chronic daily users or those in binge patterns face extended withdrawal timelines compared to people using less frequently. Pre-existing mental health conditions – depression, anxiety, PTSD – significantly amplify withdrawal severity. Environmental factors matter too: people withdrawing in environments with active drug use, social isolation, or without professional support face much higher relapse risk than those with stable environments and support systems.

Recovery and Brain Healing – The Longer Timeline That Actually Matters

If withdrawal is the first challenge, brain recovery is the marathon. The encouraging news is that the brain shows remarkable capacity for healing with sustained abstinence, even after chronic methamphetamine use.

Six Months of Abstinence – Initial Recovery Window

Studies examining cognitive function during abstinence show meaningful improvement by six months of continuous sobriety. Memory function, executive function, and processing speed – the cognitive domains most severely impaired by meth – begin showing measurable recovery. Neuroimaging studies reveal that dopamine transporter (DAT) density begins increasing after six months abstinence, indicating partial restoration of dopamine system function. Not everyone experiences complete anhedonia resolution by this point – some individuals still struggle with pleasure and motivation – but many report returning capacity to enjoy things and feel motivated, even if incompletely. The mechanisms underlying this early recovery involve neuroplasticity: the brain upregulates dopamine production, increases receptor density, and establishes new neural connections to compensate for meth-induced damage.

One Year of Abstinence – Significant Functional Recovery

By one year of continuous sobriety, cognitive function shows substantial improvement, with many long-term abstainers performing comparably to healthy controls on standardized neuropsychological testing. Dopamine system markers continue recovering, with dopamine transporter and D2 receptor density showing meaningful improvements toward normal ranges. Executive function – which depends on prefrontal cortex integrity – shows particular improvement, suggesting that the brain’s decision-making and impulse control systems are healing. Interestingly, cerebellar recovery (restoration of cerebellar brain volume) correlates positively with prolonged abstinence: the longer someone remains abstinent, the more cerebellar volume recovery occurs, paralleling behavioral improvements in motor coordination and balance.

Two Years and Beyond – Near-Complete Recovery

Individuals with two or more years of continuous abstinence often show near-complete resolution of the cognitive impairments that characterized their active use. Memory function, attention, processing speed, and executive function approach or equal levels seen in healthy, non-using controls. Brain structural changes show continued improvement, with gray matter volume recovering toward normal in previously damaged regions. The trajectory of recovery isn’t always linear – some cognitive functions recover quickly while others recover more slowly or incompletely. For example, certain memory deficits can persist longer than executive function improvements. However, the overall pattern for individuals with sustained abstinence is consistent, measurable improvement in both cognitive function and brain neurochemistry.

What Speeds Recovery – Active Engagement Matters

Recovery isn’t just about time passing. Active engagement in recovery activities – exercise, therapy, cognitive engagement, learning new skills, social connection – actively promotes neuroplasticity and accelerates brain healing beyond simple abstinence alone. This is why behavioral treatment combined with sustained abstinence produces better outcomes than abstinence alone: the combination actively stimulates brain healing through multiple pathways.

Practical Context – What Detection Windows Actually Tell Us

For people dealing with workplace drug testing, court-ordered screening, or treatment program entry requirements, understanding detection windows has practical importance. But it’s worth contextualizing what these windows actually mean.

Employment and Workplace Testing

Employers typically use urine drug testing with federal workplace cutoff levels of 500 ng/mL for initial screening and 250 ng/mL for confirmation via GC-MS. For occasional meth users, detection typically spans 2-4 days. Regular users might test positive for 5-7 days or longer. One important reality: no legally safe method exists to significantly accelerate meth clearance. Excessive water consumption, specialized diets, or commercial “detox” products are ineffective, and attempting to use them in a testing context may raise suspicion. The body’s elimination process runs on its own timeline.

Court-Ordered and Legal Screening

Court-ordered drug testing during probation or drug court participation might use urine testing, hair testing, or sweat patches depending on monitoring requirements. Hair testing (90-day window) is increasingly common in legal contexts because it identifies use patterns rather than just recent use, and it’s difficult to falsify compared to urine samples. Understanding that these longer windows catch patterns means that occasional use during probation is more likely to be detected on hair testing than it would be on urine testing.

Treatment Program Entry and Ongoing Monitoring

Most addiction treatment programs use urine testing to verify abstinence and guide treatment planning. The key reality: a negative urine test indicates absence of detectable drug but absolutely does not indicate recovery. Recovery is measured by behavioral change, sustained abstinence through difficult withdrawal periods, psychological healing, and reestablishment of healthy functioning – not by negative drug tests. Someone can test negative and still be in early withdrawal with severe cravings and psychological distress.

The Bottom Line – Detection Versus Recovery

Methamphetamine stays detectable in your system anywhere from 24-48 hours in blood to 90 days in hair, depending on the test used. For most people facing standard urine testing, expect detection for 2-4 days with occasional use, extending to 5-7+ days with regular use. The drug’s 10-12 hour half-life means it clears the bloodstream within roughly 50-75 hours for most people, though urine detection extends longer as the kidneys slowly filter out metabolites.

But the detection timeline is almost irrelevant to recovery. While meth becomes undetectable within days, withdrawal symptoms persist for weeks and neurological recovery continues for months to years. The crash phase (24-48 hours) brings overwhelming fatigue and depression. Acute withdrawal (days 3-14) brings peak cravings and significant psychological symptoms. Protracted withdrawal (weeks 2-12 and beyond) brings variable psychological symptoms that gradually improve. Brain recovery shows measurable improvement by six months of abstinence, substantial recovery by one year, and continued improvement for 1-2 years of sobriety.

This is why treatment matters more than test results. The difference between someone who successfully navigates withdrawal and remains abstinent versus someone who relapses often comes down to support, clinical oversight, and evidence-based behavioral intervention during those first difficult weeks – not whether they understand detection windows. Professional treatment helps people survive the acute phase when discomfort is worst and relapse risk is highest. That survival period is what predicts long-term recovery, and that’s where the real work happens.

Research References

1. Volkow ND, et al. Distribution and Pharmacokinetics of Methamphetamine in the Human Body: Clinical Implications. PLoS One. 2010 Dec 7;5(12):e15269. Peer-reviewed PET imaging study of methamphetamine distribution and organ-specific clearance rates in humans.
2. Cruickshank CC, Dyer KR. A review of the clinical pharmacology of methamphetamine. Addiction. 2009 Jul;104(7):1085-99. Peer-reviewed clinical pharmacology review of methamphetamine metabolism and clearance.
3. Fowler JS, et al. Fast Uptake and Long-Lasting Binding of Methamphetamine in the Human Brain. J Nucl Med. 2008 Dec;49(12):1968-74. PET imaging study of methamphetamine pharmacokinetics in the human brain.
4. Molecular, Behavioral, and Physiological Consequences of Methamphetamine Neurotoxicity: Implications for Treatment. Psychopharmacology. 2016. Comprehensive peer-reviewed review of methamphetamine neurotoxicity mechanisms and clinical manifestations.
5. Kim B, et al. Methamphetamine-Induced Neuronal Damage: Neurotoxicity and Neuroinflammation. Biomol Ther (Seoul). 2020 Jul;28(5):381-388. Peer-reviewed research on molecular mechanisms of meth neurotoxicity including oxidative stress, dopamine dysregulation, and apoptosis.
6. Alteration of Brain Structure With Long-Term Abstinence of Methamphetamine by Voxel-Based Morphometry. Frontiers in Psychiatry. 2018 Dec 19;9:722. Neuroimaging study of brain structural recovery with abstinence from methamphetamine.
7. The effects of methamphetamine intoxication on acute driving performance. Neurotoxicology. 2025 Jun. Recent research on acute neurological effects of methamphetamine.
8. Effects of acute and chronic systemic methamphetamine on autonomic cardiovascular regulation. Pharmacol Biochem Behav. 2016 Jan;140:1-11. Research on cardiovascular effects of acute and chronic methamphetamine use.
9. Current and Emerging Treatments for Methamphetamine Use Disorder. Frontiers in Psychiatry. 2021 Dec 12;12:788186. Peer-reviewed review of evidence-based behavioral and pharmacological treatments for methamphetamine use disorder.
10. University of Washington Alcohol & Drug Abuse Institute. Effective Treatments for Methamphetamine Use Disorder. 2018. Clinical guidance on contingency management, cognitive-behavioral therapy, and other evidence-based approaches.

Need Support With Methamphetamine Addiction?

Still Detox provides medical detoxification and comprehensive addiction treatment in Boca Raton, Florida. If you’re considering recovery or navigating the early stages of withdrawal, evidence-based treatment makes a real difference.

Call: (561) 556-2677